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Oral Magnetoelectric Neuroimmunomodulation via the Gut-Brain-Spleen-Heart Axis for Noninvasive Atrial Fibrillation Therapy
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Oral Magnetoelectric Neuroimmunomodulation via the Gut-Brain-Spleen-Heart Axis for Noninvasive Atrial Fibrillation Therapy

Cam-Hoa Mac, Chen-Hsuan Kuo, I-Wei Wang, Yu-Jung Lin, Cheng-Yueh Lin, Hong-Nhung Nguyen, Ninh-Son Pham, Shih-Kai Lo, Wen-Wei Wu, Hua-Jing Huang, …
Advanced materials (Weinheim), 卷.38(15), e16956
01/03/2026
PMID: 41645849

摘要

Administration, Oral Animals Atrial Fibrillation - physiopathology Atrial Fibrillation - therapy Barium Compounds - chemistry Gastrointestinal Tract Heart - physiopathology Male Neuroimmunomodulation Polymers - chemistry Polystyrenes - chemistry Rats Rats, Sprague-Dawley Spleen Thiophenes - chemistry Titanium Vagus Nerve Stimulation - methods Brain
Atrial fibrillation (AF), the most common cardiac arrhythmia, is closely associated with autonomic imbalance and inflammation. While low-level vagus nerve stimulation (LL-VNS) offers therapeutic benefits, its clinical application is limited by the need for surgical implants. Here, we present a noninvasive, orally delivered magnetoelectric neuroimmunomodulation platform that targets the gut-brain-spleen-heart axis to deliver LL-VNS for AF therapy. The platform comprises ingestible microparticles (MPs) embedding magnetostrictive Fe O @BaTiO nanogenerators (FO@BTO NGs) within a conductive matrix of poly(3,4-ethylenedioxythiophene):poly(styrenesulfonate) (PEDOT:PSS), which facilitates electrical signal transmission, and dopamine-alginate, which promotes gastric adhesion via catechol-mediated interactions. After oral gavage in a rat AF model, MPs adhere to the stomach lining and, upon alternating magnetic activation, FO@BTO NGs generate pulsed electrical currents that stimulate vagal afferent fibers. This activates brainstem autonomic circuits, enhancing parasympathetic tone and suppressing sympathetic activity. Concurrently, it engages the splenic neuroimmune circuit via the cholinergic anti-inflammatory pathway, thereby reducing systemic inflammation. This dual neuroimmune modulation significantly shortens AF duration and mitigates cardiac inflammation and electrical remodeling. These findings establish a clinically translatable, implant-free strategy for delivering LL-VNS through coordinated neural and immune pathways, offering a safe and accessible alternative to implantable systems for AF management.

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